Abstract
The protein network protecting the stability of the genome is defective in Fanconi anemia (FA). The newest in a series of FA proteins is involved in DNA damage response, but the mechanism is still unclear. Clues may come from yeast two-hybrid experiments, an extraordinarily successful tool for determining molecular function.
| Original language | English (US) |
|---|---|
| Pages (from-to) | 237-239 |
| Number of pages | 3 |
| Journal | Trends in Molecular Medicine |
| Volume | 7 |
| Issue number | 6 |
| DOIs | |
| State | Published - 2001 |
ASJC Scopus subject areas
- Molecular Medicine
- Molecular Biology