The protein network protecting the stability of the genome is defective in Fanconi anemia (FA). The newest in a series of FA proteins is involved in DNA damage response, but the mechanism is still unclear. Clues may come from yeast two-hybrid experiments, an extraordinarily successful tool for determining molecular function.
|Original language||English (US)|
|Number of pages||3|
|Journal||Trends in Molecular Medicine|
|State||Published - 2001|
ASJC Scopus subject areas
- Molecular Medicine
- Molecular Biology