Abstract
Passive cigarette smoking is becoming increasingly implicated in upper respiratory tract infections. Some 9 million to 12 million children have significant exposure to cigarette smoke, which may result in pathologic changes in mucociliary transport and cellular composition of the respiratory tract. The physiologic changes related to chronic smoke exposure have been implicated as a risk factor for otitis media and bronchitis. Currently, the cellular effects of cigarette smoke or mucosa can only be reversed or eliminated with avoidance. The use of intranasal steroids or cromolyn sodium, in addition to antibiotic therapy, when an infection is present may reduce the severity or incidence of disease.
| Original language | English (US) |
|---|---|
| Pages (from-to) | 1032-1035 |
| Number of pages | 4 |
| Journal | The Journal of allergy and clinical immunology |
| Volume | 81 |
| Issue number | 5 PART 2 |
| DOIs | |
| State | Published - 1988 |
| Externally published | Yes |
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ASJC Scopus subject areas
- Immunology
- Immunology and Allergy
Cite this
Upper airway complications of cigarette smoking. / Richardson, Mark A.
In: The Journal of allergy and clinical immunology, Vol. 81, No. 5 PART 2, 1988, p. 1032-1035.Research output: Contribution to journal › Article
}
TY - JOUR
T1 - Upper airway complications of cigarette smoking
AU - Richardson, Mark A.
PY - 1988
Y1 - 1988
N2 - Passive cigarette smoking is becoming increasingly implicated in upper respiratory tract infections. Some 9 million to 12 million children have significant exposure to cigarette smoke, which may result in pathologic changes in mucociliary transport and cellular composition of the respiratory tract. The physiologic changes related to chronic smoke exposure have been implicated as a risk factor for otitis media and bronchitis. Currently, the cellular effects of cigarette smoke or mucosa can only be reversed or eliminated with avoidance. The use of intranasal steroids or cromolyn sodium, in addition to antibiotic therapy, when an infection is present may reduce the severity or incidence of disease.
AB - Passive cigarette smoking is becoming increasingly implicated in upper respiratory tract infections. Some 9 million to 12 million children have significant exposure to cigarette smoke, which may result in pathologic changes in mucociliary transport and cellular composition of the respiratory tract. The physiologic changes related to chronic smoke exposure have been implicated as a risk factor for otitis media and bronchitis. Currently, the cellular effects of cigarette smoke or mucosa can only be reversed or eliminated with avoidance. The use of intranasal steroids or cromolyn sodium, in addition to antibiotic therapy, when an infection is present may reduce the severity or incidence of disease.
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UR - http://www.scopus.com/inward/citedby.url?scp=0023939505&partnerID=8YFLogxK
U2 - 10.1016/0091-6749(88)90175-3
DO - 10.1016/0091-6749(88)90175-3
M3 - Article
C2 - 3286730
AN - SCOPUS:0023939505
VL - 81
SP - 1032
EP - 1035
JO - Journal of Allergy and Clinical Immunology
JF - Journal of Allergy and Clinical Immunology
SN - 0091-6749
IS - 5 PART 2
ER -