Abstract
Alcohol withdrawal syndrome is characterized by hyperactivity of the nervous system. This hyperactivity represents the brain's attempt to function normally despite the inhibitory effect of chronic alcohol consumption. The syndrome manifests when alcohol consumption ceases. Experimental, clinical, and genetic research have linked the development of withdrawal to alterations in the sensitivity of neuronal communication systems. Early treatment of the syndrome is advised, because the symptom severity may increase with each subsequent episode.
| Original language | English (US) |
|---|---|
| Pages (from-to) | 149-156 |
| Number of pages | 8 |
| Journal | Alcohol Research and Health |
| Volume | 21 |
| Issue number | 2 |
| State | Published - Dec 1 1997 |
Keywords
- AOD withdrawal syndrome
- Animal model
- Animal strains
- Biological inhibition
- Chronic AODE (alcohol and other drug effects)
- Genetic mapping
- Membrane channel
- Neurochemistry
- Neurotransmission
- Neurotransmitter receptors
- Neurotransmitters
- Symptom
ASJC Scopus subject areas
- Medicine (miscellaneous)
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Cite this
Finn, D. A. (1997). Exploring alcohol withdrawal syndrome. Alcohol Research and Health, 21(2), 149-156.