Abstract
Alcohol withdrawal syndrome is characterized by hyperactivity of the nervous system. This hyperactivity represents the brain's attempt to function normally despite the inhibitory effect of chronic alcohol consumption. The syndrome manifests when alcohol consumption ceases. Experimental, clinical, and genetic research have linked the development of withdrawal to alterations in the sensitivity of neuronal communication systems. Early treatment of the syndrome is advised, because the symptom severity may increase with each subsequent episode.
| Original language | English (US) |
|---|---|
| Pages (from-to) | 149-156 |
| Number of pages | 8 |
| Journal | Alcohol Research and Health |
| Volume | 21 |
| Issue number | 2 |
| State | Published - 1997 |
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Keywords
- Animal model
- Animal strains
- AOD withdrawal syndrome
- Biological inhibition
- Chronic AODE (alcohol and other drug effects)
- Genetic mapping
- Membrane channel
- Neurochemistry
- Neurotransmission
- Neurotransmitter receptors
- Neurotransmitters
- Symptom
ASJC Scopus subject areas
- Medicine (miscellaneous)
Cite this
Exploring alcohol withdrawal syndrome. / Finn, Deborah (Deb).
In: Alcohol Research and Health, Vol. 21, No. 2, 1997, p. 149-156.Research output: Contribution to journal › Article
}
TY - JOUR
T1 - Exploring alcohol withdrawal syndrome
AU - Finn, Deborah (Deb)
PY - 1997
Y1 - 1997
N2 - Alcohol withdrawal syndrome is characterized by hyperactivity of the nervous system. This hyperactivity represents the brain's attempt to function normally despite the inhibitory effect of chronic alcohol consumption. The syndrome manifests when alcohol consumption ceases. Experimental, clinical, and genetic research have linked the development of withdrawal to alterations in the sensitivity of neuronal communication systems. Early treatment of the syndrome is advised, because the symptom severity may increase with each subsequent episode.
AB - Alcohol withdrawal syndrome is characterized by hyperactivity of the nervous system. This hyperactivity represents the brain's attempt to function normally despite the inhibitory effect of chronic alcohol consumption. The syndrome manifests when alcohol consumption ceases. Experimental, clinical, and genetic research have linked the development of withdrawal to alterations in the sensitivity of neuronal communication systems. Early treatment of the syndrome is advised, because the symptom severity may increase with each subsequent episode.
KW - Animal model
KW - Animal strains
KW - AOD withdrawal syndrome
KW - Biological inhibition
KW - Chronic AODE (alcohol and other drug effects)
KW - Genetic mapping
KW - Membrane channel
KW - Neurochemistry
KW - Neurotransmission
KW - Neurotransmitter receptors
KW - Neurotransmitters
KW - Symptom
UR - http://www.scopus.com/inward/record.url?scp=0005518917&partnerID=8YFLogxK
UR - http://www.scopus.com/inward/citedby.url?scp=0005518917&partnerID=8YFLogxK
M3 - Article
C2 - 15704352
AN - SCOPUS:0005518917
VL - 21
SP - 149
EP - 156
JO - Alcohol research : current reviews
JF - Alcohol research : current reviews
SN - 2168-3492
IS - 2
ER -