Endocannabinoids Mediate Synaptic Plasticity at Mixed Synapses

Jozsef Vigh; Henrique von Gersdorff

doi:10.1016/j.neuron.2007.12.005

Endocannabinoids Mediate Synaptic Plasticity at Mixed Synapses

Jozsef Vigh, Henrique von Gersdorff

Vollum Institute

Research output: Contribution to journal › Short survey › peer-review

3 Scopus citations

Abstract

Endocannabinoids are generally known to suppress excitatory or inhibitory synaptic transmission. Now, in an elegant series of experiments, Cachope et al. reveal a novel signaling pathway whereby endocannabinoids indirectly potentiate mixed chemical and electrical synapses. Gap junction-mediated transmission can thus be potentiated via distinct frequency-dependent mechanisms.

Original language	English (US)
Pages (from-to)	945-946
Number of pages	2
Journal	Neuron
Volume	56
Issue number	6
DOIs	https://doi.org/10.1016/j.neuron.2007.12.005
State	Published - Dec 20 2007

ASJC Scopus subject areas

General Neuroscience

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10.1016/j.neuron.2007.12.005

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abstract = "Endocannabinoids are generally known to suppress excitatory or inhibitory synaptic transmission. Now, in an elegant series of experiments, Cachope et al. reveal a novel signaling pathway whereby endocannabinoids indirectly potentiate mixed chemical and electrical synapses. Gap junction-mediated transmission can thus be potentiated via distinct frequency-dependent mechanisms.",

author = "Jozsef Vigh and {von Gersdorff}, Henrique",

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AB - Endocannabinoids are generally known to suppress excitatory or inhibitory synaptic transmission. Now, in an elegant series of experiments, Cachope et al. reveal a novel signaling pathway whereby endocannabinoids indirectly potentiate mixed chemical and electrical synapses. Gap junction-mediated transmission can thus be potentiated via distinct frequency-dependent mechanisms.

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Endocannabinoids Mediate Synaptic Plasticity at Mixed Synapses

Abstract

ASJC Scopus subject areas

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