Effect of nitric oxide synthase inhibition on the cerebral vascular response to hypercapnia in primates

Robert W. McPherson, Jeffrey R. Kirsch, Ramsis F. Ghaly, Richard J. Traystman

Research output: Contribution to journalArticle

53 Scopus citations


Background and Purpose The role of nitric oxide in cerebrovascular response to changes in PCO2 is unclear. In the present study, we assessed responses at two levels of hypercapnia in a primate model before and after blockade of nitric oxide synthesis.

Original languageEnglish (US)
Pages (from-to)682-687
Number of pages6
Issue number4
StatePublished - Apr 1995


ASJC Scopus subject areas

  • Clinical Neurology
  • Cardiology and Cardiovascular Medicine
  • Advanced and Specialized Nursing

Cite this