Blockade of JAK2-mediated extrinsic survival signals restores sensitivity of CML cells to ABL inhibitors

Elie Traer, R. MacKenzie, J. Snead, Anupriya Agarwal, A. M. Eiring, T. O'Hare, Brian Druker, M. W. Deininger

Research output: Contribution to journalArticle

65 Citations (Scopus)
Original languageEnglish (US)
Pages (from-to)1140-1143
Number of pages4
JournalLeukemia
Volume26
Issue number5
DOIs
StatePublished - May 2012

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Oncogene Proteins v-abl
Janus Kinase 2
K562 Cells
Leukemia, Myelogenous, Chronic, BCR-ABL Positive
Antineoplastic Agents

ASJC Scopus subject areas

  • Hematology
  • Cancer Research
  • Anesthesiology and Pain Medicine

Cite this

Blockade of JAK2-mediated extrinsic survival signals restores sensitivity of CML cells to ABL inhibitors. / Traer, Elie; MacKenzie, R.; Snead, J.; Agarwal, Anupriya; Eiring, A. M.; O'Hare, T.; Druker, Brian; Deininger, M. W.

In: Leukemia, Vol. 26, No. 5, 05.2012, p. 1140-1143.

Research output: Contribution to journalArticle

Traer, Elie ; MacKenzie, R. ; Snead, J. ; Agarwal, Anupriya ; Eiring, A. M. ; O'Hare, T. ; Druker, Brian ; Deininger, M. W. / Blockade of JAK2-mediated extrinsic survival signals restores sensitivity of CML cells to ABL inhibitors. In: Leukemia. 2012 ; Vol. 26, No. 5. pp. 1140-1143.
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