TY - JOUR
T1 - Acute pressure changes and possible secondary tissue changes due to laser or xenon photocoagulation
AU - McNair, James
AU - Fraunfelder, Frederick T.
AU - Wilson, R. Sloan
AU - Viernstein, Lawrence J.
AU - Babcock, Calvin
N1 - Funding Information:
From the Department of Ophthalmology, University of Arkansas Medical Center, and the Veterans Administration Hospital, Little Rock, Arkansas, and Applied Physics Laboratory (Dr. Viernstein), Johns Hopkins University, Silver Spring, Maryland. This investigation was supported in part by a grant from the National Society for the Prevention of Blindness, Inc., made possible by a contribution from the Wisconsin Council of the Blind, Inc.; and Public Health Service research grant NB 072261 from the National Institute of Neurological Diseases and Blindness. Reprint requests to F. T. Fraunfelder, M.D., Department of Ophthalmology, University of Arkansas Medical Center, 4301 W. Markham St., Little Rock, AR 72201.
PY - 1974/1
Y1 - 1974/1
N2 - The characteristics of intraocular "explosions" secondary to photocoagulation of the iris and retina are presented. With the xenon photocoagulator acoustical transients reached peak pressures of almost 900 mm Hg with pulse rise times of 0.3 msec. No significant pressure elevations were found with the argon laser even at maximum time and energy settings. In young healthy animal eyes exposed to high pressure transients, we detected no secondary tissue damage. The results from flat preparations of the corneal endothelium, light and electron microscopy were negative for secondary tissue damage even with eyes exposed to four intraocular explosions. Despite these results, clinical data suggested that, especially in diseased tissue, intraocular explosions induced by the xenon photocoagulator have the potential to cause ocular damage; therefore, these intraocular explosions should be avoided.
AB - The characteristics of intraocular "explosions" secondary to photocoagulation of the iris and retina are presented. With the xenon photocoagulator acoustical transients reached peak pressures of almost 900 mm Hg with pulse rise times of 0.3 msec. No significant pressure elevations were found with the argon laser even at maximum time and energy settings. In young healthy animal eyes exposed to high pressure transients, we detected no secondary tissue damage. The results from flat preparations of the corneal endothelium, light and electron microscopy were negative for secondary tissue damage even with eyes exposed to four intraocular explosions. Despite these results, clinical data suggested that, especially in diseased tissue, intraocular explosions induced by the xenon photocoagulator have the potential to cause ocular damage; therefore, these intraocular explosions should be avoided.
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U2 - 10.1016/0002-9394(74)90599-6
DO - 10.1016/0002-9394(74)90599-6
M3 - Article
C2 - 4207385
AN - SCOPUS:0015950346
SN - 0002-9394
VL - 77
SP - 13
EP - 18
JO - American journal of ophthalmology
JF - American journal of ophthalmology
IS - 1
ER -