Acute neurologic decompensation in an infant with cobalamin deficiency exposed to nitrous oxide

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Abstract

After exposure to nitrous oxide, an infant with unrecognized cobalamin deficiency developed neurologic deterioration and pancytopenia. Hematologic recovery and partial resolution of his neurologic changes followed repletion of the vitamin. Nitrous oxide depletes bioavailable cobalamin and may be a dangerous anesthetic in patients with cobalamin deficiency.

Original languageEnglish (US)
Pages (from-to)427-428
Number of pages2
JournalJournal of Pediatrics
Volume137
Issue number3
DOIs
Publication statusPublished - 2000

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ASJC Scopus subject areas

  • Pediatrics, Perinatology, and Child Health

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