Acute neurologic decompensation in an infant with cobalamin deficiency exposed to nitrous oxide

Research output: Contribution to journalArticle

36 Citations (Scopus)

Abstract

After exposure to nitrous oxide, an infant with unrecognized cobalamin deficiency developed neurologic deterioration and pancytopenia. Hematologic recovery and partial resolution of his neurologic changes followed repletion of the vitamin. Nitrous oxide depletes bioavailable cobalamin and may be a dangerous anesthetic in patients with cobalamin deficiency.

Original languageEnglish (US)
Pages (from-to)427-428
Number of pages2
JournalJournal of Pediatrics
Volume137
Issue number3
DOIs
StatePublished - 2000

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Nitrous Oxide
Vitamin B 12
Nervous System
Pancytopenia
Vitamins
Anesthetics

ASJC Scopus subject areas

  • Pediatrics, Perinatology, and Child Health

Cite this

Acute neurologic decompensation in an infant with cobalamin deficiency exposed to nitrous oxide. / Felmet, Kathryn; Robins, Berklee; Tilford, David; Hayflick, Susan.

In: Journal of Pediatrics, Vol. 137, No. 3, 2000, p. 427-428.

Research output: Contribution to journalArticle

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